Just The Facts - Lillian's Arrival in Medical Speak

Lillian suffered severe Hypoxic-Ischemic Encephalopathy due to anoxia at birth. The Anoxia was due to a rupture to the uterus during labor. The uterine rupture caused an intra-abdominal birth and possible abruption of the placenta. Even if the placenta did not tear, the now contracting uterus would have strangled the umbilical cord, thus cutting off the blood flow from mom to baby. Without proper blood flow, Lillian would have started starving for oxygen as the CO2 levels in her blood reached critical levels. This would cause her to try and breathe to inhale some oxygen. Unfortunately, she would now be inside her motherís abdomen, and surrounded by blood from the tear. When she inhaled, she essentially drowned in a combination of hers and Sallyís blood. The CO2 levels in her blood now reached lethal levels, and she died. When she was delivered, she had an initial APGAR score of 0. The doctor had resuscitated her at one minute. That is, the doctor restarted her heart, and had her successfully intubated. At five minutes, her APGAR was only up to 2, and at 10 minutes it was only 3. She still could not breathe on her own, and was transported to a Level 3 ICU facility. Her blood gas levels at birth were not compatible with life. The transport nurse who picked her up in the Life-Flight helicopter told me she thought she would be picking up a corpse. Her O2 (Oxygen) level was only 7. It should be in the 700 or 800 range. Her CO2 (Carbon Dioxide) level was 147. It should be in the 40s. Her overall Ph level was not what you would find in a live infant. Her pupils were dilated and unresponsive. Her reflexes were non-existent. The only positive sign of life was the fact that after resuscitation her heart continued to beat strongly.